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Brain Storming

Most of the perfusion to coronary artery occurs during:

  1. Ventricular systole
  2. Atrial systole
  3. Ventricular diastole
  4. Atrial relaxation
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Ventricular diastole


Increases incidence of acute coronary syndromes is not associated with:

  1. Smoking
  2. Morbid obesity
  3. Diabetes
  4. Type A personality
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Type A personality


Ist, 2nd and third heart blocks are first discovered by:

  1. Wenkebach
  2. Einthoven
  3. Mobitz
  4. Bachman
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Wenkebach


The three lead system I, II, & III was developed by:

  1. Wenkebach
  2. Einthoven
  3. Mobitz
  4. Kent
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Einthoven


The rhythm  associated with sudden cardiac death is:

  1. VF
  2. VT
  3. Ventricular escape
  4. Sinus bradycardia below 20 bps
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VF


Average size of ventricle compared to atria is:

  1. Same size
  2. Two times larger
  3. Three times larger
  4. Four times larger
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Three times larger


In patients with IHD,  the following is associated with less incidence of AMI:

  1. Calcium channels blockers
  2. GTN
  3. Beta blockers
  4. Aspirin
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Aspirin


Anginal pain is described as:

  1. Stabbing
  2. Crushing
  3. Tearing
  4. Stinging
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Crushing


The speed of an ECG strip is usually:

  1. 25 mm/sec
  2. 50 mm/sec
  3. 10 mm/sec
  4. 5 mm/sec
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25mm/sec


Greatest chance of adult survival from VF is associated with:

  1. Prompt CPR with pushing hard and fast
  2. Adrenalin 1mg IV every 4 minutes during CPR
  3. Early Defibrillation
  4. 100% oxygen administration during CPR
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Early Defibrillation


Cardiac Output in adults is:

  1. 2-3 l/min
  2. 7-10 l/min
  3. 5-6 l/min
  4. Normally >10 l/m
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5-6 l/min


Which medication is not given routinely in acute MI?

  1. Beta blockers
  2. Lidocaine (as an antiarrhythmic)
  3. Nitroglycerine
  4. Aspirin
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Lidocaine


The most common complication associated with thrombolytics is:

  1. Anaphylaxis
  2. Arrhythmias
  3. Stroke
  4. Bleeding
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Bleeding


Tenecteplase is:

  1. An anticoagulant
  2. A thrombolytic agent
  3. An anti-platelet
  4. Glycoprotein IIb/IIIa inhibitor
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A thrombolytic agent


An occlusion of the R coronary artery most likely causes:

  1. An inferior MI
  2. An anterior MI
  3. A posterior MI
  4. A lateral MI
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An inferior MI


Thrombolytic therapy in ACS is aimed at:

  1. Destroying cholesterol plaques
  2. Prevention of the enlargement of intracoronary thrombosis
  3. Preventing deep vein thrombosis
  4. Dissolving the intracoronary thrombus
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Dissolving the intracoronary thrombus


 

In unstable angina Beta Blockers may reduce the risk of:

  1. Stroke
  2. Pulmonary oedema
  3. MI
  4. DVT
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MI


The S3 heart sound is associated with:

  1. Congestive heart failure
  2. Stroke
  3. Low cardiac output
  4. Junctional rhythm
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Congestive heart failure


The S4 heart sound is often associated with:

  1. Pulmonary hypertension
  2. Fibrosis of the ventricle following MI
  3. Unstable angina
  4. Austin Flint murmur
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Fibrosis of the ventricle following MI


Presence of ventricular ectopics with coexisting chest pain perhaps best managed initially with:

  1. Lidocain infusion
  2. Amiodarone infusion
  3. Magnesium sulphate 2g slow IV
  4. Supplementary Oxygen
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Supplementary Oxygen


Starling Low states that:

  1. Increased stretch of cardiac muscle fibers results in increased force of contractions
  2. Increased after load leads to reduction of C.O.
  3. Reduced preload reduces C.O.
  4. None of above statements.
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Increased stretch of cardiac muscle fibers results in increased force of contractions


Symptom often associated with descending aortic dissection:

  1. Tearing mid scapular pain
  2. Crushing chest pain
  3. Bradycardia
  4. Coma
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Tearing mid scapular pain

The location of the pain may indicate the site of dissection. Anterior chest pain and chest pain that mimics acute myocardial infarction usually are associated with anterior arch or aortic root dissection. This is caused by the dissection interrupting flow to the coronary arteries, resulting in myocardial ischemia. Pain in the neck or jaw indicates that the dissection involves the aortic arch and extends into the great vessels.

Tearing or ripping pain in the intrascapular area may indicate that the dissection involves the descending aorta. The pain typically changes as the dissection evolves.

The pain of aortic dissection is typically distinguished from the pain of acute myocardial infarction by its abrupt onset and maximal severity at onset, although the presentations of both conditions overlap to some degree and are easily confused. Aortic dissection can be presumed in patients with symptoms and signs suggestive of myocardial infarction but without classic electrocardiographic (ECG) findings.


The following is not usually associated with Atrial fibrillation:

  1. Congestive heart failure
  2. Hypertension
  3. Ventricular fibrillation
  4. Rheumatic Heart Disease
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Ventricular fibrillation


Adrenaline administered in refractory VF

  1. Reduces morbidity and mortality
  2. Is an effective antiarrhythmic
  3. Increases coronary perfusion pressure during CPR
  4. Is more effective when given in higher doses and more frequently
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Increases coronary perfusion pressure during CPR


An average stroke volume during resting state is:

  1. 10-30ml
  2. 35-45ml
  3. 50-80ml
  4. 90-130 ml
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50-80ml


Which drug when given with rTPA in AMI enhances its efficacy?

  1. Beta blockers
  2. Aspirin
  3. Low molecular weight heparin
  4. Nitroglycerine
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Aspirin


The reduction in CO due to loss of atrial kick in Atrial Fibrillation is:

  1. <5%
  2. 10%
  3. 15-35%
  4. There is virtually no effect at all
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15-35%


Crushing chest pain and tightness may be a symptom of:

  1. Angina pectoris
  2. Acute pancreatitis
  3. Pulmonary embolization
  4. All of above
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All of above


The management of acute myocardial infarction is:

  1. To reduce after load to the heart
  2. Increase oxygen supply to the myocardium
  3. Increase oxygen supply and reduce demand
  4. Generally symptomatic only, as the damage already done
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Increase oxygen supply and reduce demand


The endothelium possesses an endocrine role in:

  1. Stimulating and inhibiting vessel growth
  2. Influencing arterial tone
  3. Affecting coagulation
  4. All of above
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All of above


Beta blockers should not be used in MI when associated with:

  1. Sinus tachycardia
  2. AF
  3. Mobitz type II block
  4. VT
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Mobitz type II block


The ECG criteria for AMI:

  1. ST elevation (> 1mm) in leads 1 & AVL
  2. ST elevation (> 1mm) in leads 2,3 & AVF
  3. ST elevation (> 1mm) in leads V1-V4
  4. New Q waves in 2,3 &AVF
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ST elevation (> 1mm) in leads 2,3 & AVF


Criteria for thrombolysis does not include:

  1. ST depression
  2. ST elevation in leads 1,aVl and V5-V6
  3. St elevation in leads 2,3 & aVF
  4. New LBBB associated wit ST elevation I Leads V1-V4
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ST depression


R ventricular infarct is associated with:

  1. Inferior MI
  2. Anterior MI
  3. Antero-septal MI
  4. Lateral MI
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Inferior MI


Glycoprotein 2b/3a antagonists are beneficial in:

  1. Acute pulmonary oedema
  2. Cardiogenic shock
  3. Unstable angina
  4. Stroke
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Unstable angina


High risk in acute coronary syndrome include:

  1. Elevated CKMB, angina controlled with medication an normal LV function
  2. Angina at rest with medications, elevated Troponins, ST depression and a prior MI
  3. Family history of CAD, prior use of aspirin and a normal ECG
  4. One episode of angina while on medical therapy, Normal CKMB but elevated Troponins and prior MI
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Angina at rest with medications, elevated Troponins, ST depression and a prior MI


 
    
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